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Migrenesmerte - mekanismer og konsekvenser for behandling
Engelsk titel: Migraine - mechanisms and consequences for treatment Läs online Författare: Alstadhaug KB ; Salvesen R Språk: Nor Antal referenser: 53 Dokumenttyp: Artikel UI-nummer: 07121956

Tidskrift

Tidsskrift for Den Norske Laegeforening 2007;127(23)3064-8 ISSN 0029-2001 E-ISSN 0807-7096 KIBs bestånd av denna tidskrift Denna tidskrift är expertgranskad (Peer-Reviewed)

Sammanfattning

BACKGROUND : Migraine is a common headache syndrome. The understanding of processes in the brain during a migraine attack has increased significantly during recent years. MATERIAL AND METHODS : The article is based on literature retrieved from PubMed, and on own knowledge of older and more recent migraine literature. RESULTS AND INTERPRETATION : Patients with migraine probably have increased cortical excitability. Attacks may be initiated by the neurophysiological phenomenon "cortical spreading depression". This may again lead to meningeal inflammation and irritation of pain-sensitive fibres in the trigeminal nerve. There is compelling evidence that an episodic dysfunction of the brain stem plays an important role, either as a primary generator of or a secondary contributor to, migraine attacks. Patients often report allodynia as an expression for central sensitization. Activity in pain-modulating mechanisms is disturbed, and serotonin agonists efficiently stop attacks. The tendency to have repetitive migraine attacks is probably due to a genetic predisposition.