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Influence of ABCB1 polymorphisms and serum concentrations on venlafaxine response in patients with major depressive disorder
Engelsk titel: Influence of ABCB1 polymorphisms and serum concentrations on venlafaxine response in patients with major depressive disorder Läs online Författare: Ozbey, Gul ; Celikel, Feryal Cam ; Cumurcu, Birgul Elbozan ; Kan, Derya ; Yucel, Berna ; Hasbek, Ekrem ; Percin, Ferda ; Guzey, Ismail Cüneyt ; Uluoglu, Canan Språk: Eng Antal referenser: 63 Dokumenttyp: Artikel UI-nummer: 17060135

Tidskrift

Nordic Journal of Psychiatry 2017;71(3)230-7 ISSN 0803-9488 E-ISSN 1502-4725 KIBs bestånd av denna tidskrift Denna tidskrift är expertgranskad (Peer-Reviewed)

Sammanfattning

Background: The pharmacokinetics and the pharmacodynamics of antidepressants show large interindividual variations which result in unpredictable clinical responses. Aim: The aim of the study was to examine the effect of ABCB1 polymorphisms and the serum concentrations on the efficacy and tolerability of venlafaxine in patients with major depressive disorder (MDD). Methods: Fifty-two outpatients who met the Diagnostic and Statistical Manual of Mental Disorders Fourth Edition (DSM-IV) criteria for MDD were recruited for the study. The severity of depression was assessed using the 17-item Hamilton Rating Scale for Depression scale (HDRS17) and tolerability was assessed based on a query regarding side-effects for 6 weeks. The ABCB1 C3435T/A and G2677T/A polymorphisms were genotyped by PCR/RFLP and steady-state serum venlafaxine concentrations were measured by high-performance liquid chromatography. Results: Patients with the TT genotype for the C3435T and the TT/TA genotype for the G2677T/A polymorphism showed significantly higher frequencies in venlafaxine-induced akathisia. This relationship was not observed for efficacy. As regards serum venlafaxine concentrations, patient groups showed no significant differences in efficacy and tolerability. Conclusion: The results suggest that individuals with the TT-TT/TA genotypes for the C3435T-G2677T/A polymorphisms of ABCB1 may be pre-disposed to a risk of akathisia.